Cisplatin is a major anticancer drug that kills cancer cells by damaging their DNA and inducing apoptosis. Cisplatin is a small and remarkably simple molecule composed of one platinum atom linked to two amides and two chlorides. In its reactive form (with two H₂O molecules replacing two chlorines), cisplatin covalently binds to DNA bases, particularly purines, forming DNA adducts on the same strand (intrastrand) or on the opposite strands (interstrand) to distort the helix.
What repair mechanism would repair the bulky DNA adducts or lesions generated by cisplatin?

A) Nucleotide Excision Repair (NER)
B) Non-Homologous End Joining (NHEJ)
C) Mismatch Repair (MMR)
D) Homologous Recombination (HR)